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However, when people with allergic rhinitis have other coexisting disorders, such as asthma, the disease is much more serious, with rare cases of death. Although the condition is not considered serious (if there is no asthma), it causes a lot of discomfort and interferes with the normal daily life of the patient.
There is no simple and quick way to treat allergic rhinitis in allergic people. What to do in each case, only a qualified doctor will tell you.
To completely get rid of the disease, you will have to undergo complex diagnostics and a long course of treatment. In the future, you will need to avoid contact with allergens and adhere to a whole set of rules.
Sometimes moving to a different climate zone dramatically helps to solve this problem.
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General practitioner, gastroenterologist, hepatologist, infectious disease specialist. I carry out preventive measures for complications from the digestive system after long-term therapy with NSAIDs and blood-thinning drugs.
Alveolar proteinosis of the lungs is a lung disease of unknown etiology, characterized by the accumulation of a protein-lipid substance in the alveoli and moderately progressive shortness of breath.
Pulmonary alveolar proteinosis was first described by Rosen et al in 1958. The disease is rare, men are ill two to three times more often than women, usually in middle age. The etiology of pulmonary alveolar proteinosis is not clear. Many associate it with the damaging effect of environmental factors (industrial gases, chemicals). The role of viral and fungal infections is discussed. immune disorders.
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There are indications of drug allergy and a correlation between immunosuppressive therapy and the development of pulmonary alveolar proteinosis. Pathogenesis of pulmonary alveolar proteinosis.
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Various assumptions have been made regarding the pathogenesis of pulmonary alveolar proteinosis: the penetration of normal plasma proteins through the alveolar-capillary membrane, followed by desquamation of the epithelium; primary changes in the integumentary epithelium of the alveoli with fatty degeneration, desquamation, necrosis and secondary dysfunction of the alveolar-capillary membrane; increased local production of immunoglobulins (in the lungs), especially immunoglobulin G and immunoglobulin A, with a violation of their catabolism due to a general defect in the removal of levitra pills from the lungs. It has now been proven that the secret in the alveoli in pulmonary alveolar proteinosis contains phospholipids and free fatty acids, is similar in composition to a surfactant, but does not have surface-active properties.
In this regard, it is believed that the disease is based on a decrease in the function of alveolar macrophages to cleanse the alveoli from surfactant and, possibly, a lack of proteolytic activity of bronchial secretions, with a special role played by the activity of Vardenafil. It is assumed that disorders of the surfactant system of the lungs in pulmonary alveolar proteinosis are genetically determined.
The alveoli of patients are filled with granular eosinophilic substance rich in lipids, with a sharply positive PAS reaction, in which there are accumulations of levitra macrophages with lipid granules in the cytoplasm. Inflammatory reaction in the interalveolar septa is not expressed. In some areas, lymphocytic infiltration around small vessels is observed.
Pathomorphology of pulmonary alveolar proteinosis. Symptoms of alveolar proteinosis of the lungs.